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316 , 317 This oxidative stress environment may further activate tau protein kinases (such as GSK-3) and accelerate the abnormal phosphorylation process of tau protein

Similarly, ATMs in lean subjects are supported by fatty acid oxidation (FAO), glycolysis and glutaminolysis (anti-inflammatory macrophage), while ATMs in obese subjects depend more on glycolysis (pro-inflammatory macrophage) [9]